Axonal swellings act as a sink for action potentials.
Alzheimer’s disease is characterized by the abnormal accumulation of two misfolded proteins — amyloid-β, which forms structures called plaques outside cells, and tau inside cells.
However, the effect of these aggregates on neuronal and network functions has been difficult to assess. The highly aggregated amyloid-β that makes up the core of plaques is surrounded by areas in which the synaptic connections between neurons have been lost
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