New research paves the way for new strategies to address long-term COVID-19 complications. Researchers from the University of Malta have identified a possible cause for the prolonged and frequently debilitating symptoms faced by individuals with long COVID-19. The recent study, which was published
Researchers have found a potential cause for long COVID-19’s enduring symptoms. Their study revealed that the ACE2 receptor, which the SARS-CoV-2 virus targets, plays a pivotal role. Reducing ACE2 levels in fruit flies resulted in fatigue and mobility issues. The findings highlight the importance of ACE2 in the neuromuscular issues seen in COVID-19 patients, offering a path for future therapeutic solutions.
Around one in three individuals who recover from COVID-19 continue to experience life-disrupting symptoms, such as persistent fatigue, shortness of breath, ‘brain fog’ , and muscle weakness. The origin of long COVID, despite its increasing global impact on daily life, has remained a mystery., the coronavirus responsible for COVID-19, latches onto the ACE2 receptor, which acts as the doorway through which theinfects cells.
“Our research clearly shows that depletion of ACE2 is central to the neuromuscular complications experienced by a significant percentage of COVID-19 patients,” said Professor Ruben Cauchi, who heads the Motor Neuron Disease Laboratory at the University of Malta. When analyzing molecular defects in organisms with downregulated ACE2 levels, the Maltese scientists discovered a breakdown in communication between nerves and muscles. Several key molecules required for nerves to send messages to muscles were found compromised.
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