Pancreatic cancer is one of the deadliest forms of cancer, with a survival rate of about 10%.
While these are all impressive feats, there has been no definite cure for pancreatic cancer. in New York has been able to zero in on a protein that kick-starts and accelerates the formation of pancreatic tumors.The team explored the role of RNA splicing, a genetic process that happens before protein production, in pancreatic cancer. The team found a protein called SRSF1, which helps regulate the process of RNA splicing.
The SRSF1 levels in the body must remain at a specific level and there’s a range of genes and proteins working together just to achieve that. But as it happens, due to numerous factors, this process can sometimes be irregular. This, unfortunately, causes inflammation, which often results in the formation of Pancreatic ductal adenocarcinoma tumors.
PDAC is not only the most highly aggressive type of cancer but also the most prevalent form of pancreatic cancer. Over 90 percent of its patients die within five years of their diagnosis.The team of scientists conducted their study in an animal model. They found that when SRSF1 returned to normal levels, the organoids stopped growing, said theCSHL Professor Adrian Krainer said, “PDAC is often found too late for treatments like chemotherapy and surgery to be very effective.
Krainer said that there is still a lot of work to be done in order to explore the benefits of this research.
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