Three antiviral drugs and a monoclonal antibody may keep people newly diagnosed with COVID-19 from becoming severely ill and dying.
have reported that some people have relapses of the virus a few days after stopping Paxlovid. No one really knows how common that is or why it happens. The U.S. National Institutes of Health are collaborating with the CDC and the FDA to better understand the phenomenon, according to a statement the National Institute of Allergy and Infectious Diseases sent to me. But the agencies have no formal studies underway.
Experts I talked to had some ideas about what might be happening. It could be that the immune system “chills out” while people are taking the drug, giving the virus the opportunity to rebound once the drug is stopped, Gallagher speculates. Or the virus might hide in certain places in the body that the drug has trouble reaching, Denison says. Both of those situations might be solved by giving people the drug for longer, they say.
Resistance to the drug is another reason infections might relapse. “I think drug resistance is the least likely explanation,” says Denison. For resistance to develop, the virus has to replicate, which it can’t do when the drug is around. Still, researchers should watch for mutations in viruses taken from relapsed patients that might indicate resistance, he says.
in people who were exposed to infected household members, but the result wasn’t statistically meaningful, Pfizer reported April 29 in a news release. That means it’s not great as a preventative and won’t affect case counts. “What [the drugs] could flatten is a surge in hospitalizations and deaths,” Gallagher says. But, he notes, people at high risk of severe disease should not rely on the drugs alone to save them. “I don’t think that any of these therapeutics is as good as a booster.”Scientists and journalists share a core belief in questioning, observing and verifying to reach the truth.reports on crucial research and discovery across science disciplines.
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